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A.GugasyanR.et al

Collectively, these findings establish ROS as critical enablers of HCC metastasis, orchestrating EMT, proteolytic matrix degradation, and distant organ colonization through coordinated activation of ERK/AKT, JNK, and NF-B signaling [91], thereby providing a mechanistic basis for the link between oxidative stress and poor clinical outcomes in HCC

Although SAMe has a favorable safety profile, it is still worth paying attention to the risk that SAMe might cause increased Hcy levels, which is associated with higher risk of cardiovascular disease [211]

The next section answers common questions and ties together the themes of this guide